Dissident AIDS Database

Co-factorsAntibioticsGeneralImmuno-deficiency
Apocalypse Now: How Antibiotics Breed Disease
 Cannon Geoffrey
  Once you know, that you need the resident bacteria in your gut to protect your health, and that antibiotics especially when overused may eventually not only devaste these friendly flora but also may strip away the outer immune defences in your gut, it is easy to see that prolonged course of antibiotics can in time lay you open to all sorts of infections and also non-infectious diseases…Our resident gut flora ... stimulate the production of immunoglobulins, proteins in the blood integral to the body's inner immune defences. Experiments show that animals with all their gut flora removed, make only about one-fiftieth as much immunoglobuline as normal animals. Commenting on this finding, the standard textbook 'Immunology' states: "If the commensal organisms of the gut are removed by antibiotics, pathogenic organisms can readily gain a foothold"… it follows inescapably that all antibiotics are by their nature immunosuppressants -mildly so, no doubt…, but immunosuppressive none the less. How much this matters depends on the general state of the health of the individual, the type of antibiotic and the strength and length of the course… More than any other community, people whose lifestyle involves very many sexual partners are almost certain to suffer combinations and permutations of sexually transmitted diseases, which when bacterial are treated with constant courses of antibiotics, often broad-spectrum and/or cocktails. Such treatment over time provokes superinfection and drug-resistant superbugs -so more antibiotics are used, often more toxic in their effect… In his book "The Plague Maker, Dr. Jeffrey Fisher states that Dr. Luc Montagnier of the Pasteur Institute in Paris, co-discoverer of HIV, believes that gross overuse of antibitocs may be a co-factor with HIV development of full-blown AIDS."
  Superbugs, Nature's Revenge, Virgin Publishing Ltd., London 19951995
Predisposing factors in Pneumocystis carinii pneumonia: effects of tetracycline, protein malnutrition, and corticosteroids on hosts
 Walzer PD, LaBine M, Redington TJ, Cushion MT
  Components of the immunosuppressive regimen used to reactivate latent Pneumocystis carinii infection were analyzed for their effects on the growth, nutrition, and lymphoid system of hosts. Rats that were administered either tetracycline or a low-protein (8%) diet alone for 7 weeks developed few abnormalities, but animals on the combined regimen developed lower body and lymphoid organ weights, lower serum albumin levels, and fewer circulating lymphocytes. Rats that were administered corticosteroids and tetracycline experienced severe wasting, debilitation, and generalized lymphocyte depletion; the low-protein diet increased the magnitude of these changes. Alterations in the frequency of occurrence of specific lymphocyte subsets occurred only in rats given corticosteroids and consisted mainly of a greater decline in peripheral blood T helper cells than in T suppressor cells. The data suggest that long-term tetracycline administration and a low-protein diet have a variety of adverse effects on the host which enhance the immunosuppressive properties of corticosteroids.
  Infect Immun 1984 Dec;46(3):747-531984