Dissident AIDS Database

NIAID paper : HIV causes AIDSAfricaEtiologyMonkey aids
  "African green monkeys do not develop AIDS despite high levels of the simian immunodeficiency virus (SIV) in their blood... Scientists have known for years that African green monkeys can be infected with SIV and not develop AIDS but it was believed that this was due to effective immune system control of the virus, which would appear as a low viral load in the blood. S. Broussard and colleagues, though, found high levels of the virus in the blood of all the infected monkeys ("Simian immunodeficiency virus replicates to high levels in naturally infected African green monkeys without inducing immunologic or neurologic disease," Journal of Virology, 2001;75:2262-2275). They also found high levels of viral replication in the central nervous system, believed to be the primary cause of neurological wasting, without any symptoms in the monkeys. "These data clearly indicate that high levels of viremia and high rates of virus replication do not necessarily lead to disease and that specific host factors or the particular nature of the host response plays a critical role in determining whether disease arises following infection," said the researchers."
  AIDS Weekly 2 April 20012001
First HIV-positive lab chimp finally develops AIDS after eleven years
 Philpott Paul
  One chimp finally developed AIDS, according to scientists at the Yerkes Primate Research Center at Emory University in Atlanta. Jerom, one of 13 Yerkes chimpanzees participating in various HIV-positive primate studies at several institutions nation-wide (on a total of 125 to 150 laboratory chimps world-wide who were experimentally injected with HIV over ten years ago) developed one AIDS symptom (thrombocytopenia) in 1989, recovered, then remained healthy until March, 1995. According to a news release issued by Yerkes' Cathy Yarbrough, Jerom's CD4 immune cell [count] dropped from about one thousand to 90 prior to developing clinical symptoms : chronic-to-severe diarrhea, coughing, low-grade pneumonia, and his most dire symptom anemia, which was becoming more severe and untreatable. For this reason, Jerom was finally humanely euthanized. Jerom never received any "anti-HIV" drugs, including AZT, but was "treated for clinical diseases that occurred secondary to the HIV infection. The researchers transfused about 40 milliliters of the ailing chimp's blood into another chimpanzee [Nathan] that had never been exposed to either SIV or HIV. In just two weeks, the second chimp's CD4 levels dropped from 1,200 to 320 and eventually to 10 without Nathan developing any clinical AIDS symptoms. 1) After observing 125 to 150 HIV-positive chimps for eleven years, researchers have reported only two cases of AIDS, in Jerom and Nathan, yielding a rate of 1.6% to 1.3%. Those are remarkably low figures compared to the official view that 50% to 95% of all humans will develop AIDS within ten years of becoming HIV-positive. That eleven-year rate for AIDS conditions in HIV-positive lab chimps is so low that it is hard to imagine that it could be even lower in HIV-negative chimps (the HIV-positive chimps, who were on average are about 15 years old according to the reports, every day grew closer to the end of their natural life expectancy and were bound to start losing body mass and mental capacity, and become susceptible to opportunistic infections such as pneumonia, just like aging humans). 2) Jerom's most serious clinical symptom was anemia, which is not an AIDS condition. 3) The case of Nathan, the chimp whose T4 counts began a profound decline immediately upon being transfused with Jerom's blood is a curiosity. Nowhere in the medical literature is there a precedent for AIDS appearing so quickly upon exposure to HIV, which is why a ten year latency period is claimed for it. It is odd indeed that of the first 150 HIV-positive lab chimps, the only one to develop persistent AIDS conditions should do so in the eleventh year, but the next chimp should develop a persistent AIDS condition immediately... It is well known that blood transfusions are inherently immune suppressive.