Dissident AIDS Database

NIAID paper : HIV causes AIDSDeveloped countriesDrug-AIDS connectionIV drug abuse
The Drug-AIDS Hypothesis
 Duesberg Peter, Rasnick David
  (Margolick JB, Munoz A, Vlahov D, Solomon L, Astemborski J, Cohn S, Nelson KE. Changes in T-lymphocyte subsets in intravenous drug users with HIV-1 infection. Journal of the American Medical Association 1992; 267: 1631-1636) Based on analyses of HIV-positive intravenous drug users, "with 45% injecting at least once per day," Margolick et al. conclude "that progression of HIV-1 infection in IV drug users, as reflected in the decline of CD4 cell counts, is no more rapid than that reported for other risk groups". In an effort to exclude the role of drugs in AIDS, the authors pointed out that in a particular 6 month survey interval there was no "effect of active vs inactive drug use" on T cell loss. However, there was no verification for "inactive" drug use, and no informatiom as to whether "inactive" street drug use was substituted by methadone, which is itself immune suppressive (Klimas NG, Blaney NT, Morgan RO, Chitwood D, Milles K, Lee H, Fletcher MA. Immune Function and Anti-HTLV-I/II Status in Anti-HIV-1-Negative Intravenous Drug Users Receiving Methadone. The American Journal of Medicine 1991; 90: 163-170). Moreover there was no effort to determine the cumulative lifetime drug dose of active or "inactive" drug users that is essential to evaluate drug pathogenicity. There was also no information as to whether "other risk groups" included drug-free controls.
  Continuum Feb./March 19971997